和厚樸酚
分析標(biāo)準(zhǔn)品,HPLC≥98%
Honokiol
CAS號(hào)：35354-74-6
分子式：C18H18O2
分子量：266.33
MDL：MFCD00016674
貨號(hào) | 規(guī)格/參數(shù)/品牌 | 價(jià)格 | 貨期 |
YJ-B20498-20mg | 分析標(biāo)準(zhǔn)品,HPLC≥98% | ￥180.00 | 現(xiàn)貨 |
JS31381-1g | 98% | ￥520.00 | 現(xiàn)貨 |
JS31381-5g | 98% | ￥1280.00 | 現(xiàn)貨 |
JS31381-25g | 98% | ￥2980.00 | 現(xiàn)貨 |
A10304-20mg | 分析標(biāo)準(zhǔn)品,含量98.7%，可溯源 | ￥270.00 | 現(xiàn)貨 |
YJ-BW10003-50mg | 分析標(biāo)準(zhǔn)品,標(biāo)準(zhǔn)物質(zhì)，見(jiàn)證書(shū) | ￥294.00 | 現(xiàn)貨 |
產(chǎn)品介紹
熔點(diǎn)：87.5℃
沸點(diǎn)：400.1℃ at 760 mmHg
外觀：棕褐色至白色精細(xì)粉末，單體為無(wú)色鱗片狀晶體
溶解性：可溶于一般的有機(jī)溶劑，易溶于苯，乙mi，氯仿，乙醇等，難溶于水。
儲(chǔ)存條件：2-8℃
注意：部分產(chǎn)品我司僅能提供部分信息，我司不保證所提供信息的權(quán)威性，僅供客戶(hù)參考交流研究之用。
參考文獻(xiàn)(39篇)
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34. [IF=4.8] Haitao Li et al."Honokiol relieves hippocampal neuronal damage in Alzheimer's disease by activating the SIRT3‐mediated mitochondrial autophagy."CNS Neuroscience & Therapeutics.2024 Aug; 30(8): e14878
33. [IF=8.2] Yulong Wei et al."New insight into molecular mechanisms of different polyphenols affecting Sirtuin 3 deacetylation activity."INTERNATIONAL JOURNAL OF BIOLOGICAL MACROMOLECULES".2024 May;:132026
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28. [IF=3.251] Kanglei Zhang et al."Honokiol Antagonizes Cadmium-Induced Nephrotoxicity in Quail by Alleviating Autophagy Dysfunction, Apoptosis and Mitochondrial UPR Inhibition with Its Antioxidant Properties."Life-Basel.2022 Oct;12(10):1574
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24. [IF=1.719] Xiaoying Zhu et al."Honokiol suppresses TNF-α-induced migration and matrix metalloproteinase expression by blocking NF-κB activation via the ERK signaling pathway in rat aortic smooth muscle cells."Acta Histochem. 2014 May;116:588
23. [IF=1.902] Junjun Wang et al."The pharmacokinetics and tissue distribution of curcumin and its metabolites in mice."Biomed Chromatogr. 2018 Sep;32(9):e4267
22. [IF=2.939] Fan Shuli et al."Honokiol Inhibits Tumor Necrosis Factor-α-Stimulated Rat Aortic Smooth Muscle Cell Proliferation via Caspase- and Mitochondrial-Dependent Apoptosis."Inflammation. 2014 Feb;37(1):17-26
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18. Qiu, Ling, et al. "Honokiol ameliorates endothelial dysfunction through suppression of PTX3 expression, a key mediator of IKK/IκB/NF-κB, in atherosclerotic cell model." Experimental & molecular medicine 47.7 (2015): e171-e171.
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